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Search Results to Teresita Bellido

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One or more keywords matched the following items that are connected to Bellido, Teresita

Item TypeName
Academic Article Chronic elevation of parathyroid hormone in mice reduces expression of sclerostin by osteocytes: a novel mechanism for hormonal control of osteoblastogenesis.
Academic Article PTH receptor signaling in osteocytes governs periosteal bone formation and intracortical remodeling.
Academic Article Downregulation of SOST/sclerostin by PTH: a novel mechanism of hormonal control of bone formation mediated by osteocytes.
Academic Article Anabolic and catabolic regimens of human parathyroid hormone 1-34 elicit bone- and envelope-specific attenuation of skeletal effects in Sost-deficient mice.
Academic Article Cell autonomous requirement of connexin 43 for osteocyte survival: consequences for endocortical resorption and periosteal bone formation.
Academic Article Resorption controls bone anabolism driven by parathyroid hormone (PTH) receptor signaling in osteocytes.
Academic Article Mechanical stimulation of bone in vivo reduces osteocyte expression of Sost/sclerostin.
Academic Article Mechanical stimulation in vivo reduces osteocyte expression of sclerostin.
Academic Article Sost downregulation and local Wnt signaling are required for the osteogenic response to mechanical loading.
Concept Adaptor Proteins, Signal Transducing
Academic Article The Sirtuin1 activator SRT3025 down-regulates sclerostin and rescues ovariectomy-induced bone loss and biomechanical deterioration in female mice.
Academic Article High Bone Mass-Causing Mutant LRP5 Receptors Are Resistant to Endogenous Inhibitors In Vivo.
Academic Article Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma.
Academic Article Protection From Glucocorticoid-Induced Osteoporosis by Anti-Catabolic Signaling in the Absence of Sost/Sclerostin.
Academic Article Control of Bone Anabolism in Response to Mechanical Loading and PTH by Distinct Mechanisms Downstream of the PTH Receptor.
Academic Article Genetic deletion of Sost or pharmacological inhibition of sclerostin prevent multiple myeloma-induced bone disease without affecting tumor growth.
Academic Article PTHrP-Derived Peptides Restore Bone Mass and Strength in Diabetic Mice: Additive Effect of Mechanical Loading.
Academic Article Skeletal cell YAP and TAZ combinatorially promote bone development.
Academic Article TG-interacting factor 1 (Tgif1)-deficiency attenuates bone remodeling and blunts the anabolic response to parathyroid hormone.
Academic Article Molecular Basis for Craniofacial Phenotypes Caused by Sclerostin Deletion.

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  • Adaptor Proteins Signal Transducing